Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
➤ Gửi thông báo lỗi ⚠️ Báo cáo tài liệu vi phạmNội dung chi tiết: Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
PartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2ii>, and Aitw III Aimi'*.IIk-ú.i .111(1 flilii.il f.«r, Siuqii.il fli'iXMlint'iil, Picivinikd Hi-.tllh fail" Slimline, Rtiloqii.i, ll.dyThe postrftsuscitation syndrome (PUS) has been defined as a condition of an organism resuscitated following prolonged cardiac arrest, caused by a combination of who Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2le body ischemia and reperfusion, and characterized by multiple organ dysfunction, including neurologic impairment.1BackgroundFollowing resuscitationEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
from cardiac arrest, patients either recover consciousness or remain unconscious, depending on the duration of cardiac arrest and the effectiveness ofPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2usion and promote the restoration of spontaneous circulation (c.g.. bystander CPK, early deflbril lalion. and other means) improves the possibility of a successful outcome with the patient recovering consciousness.3Hie wider availability of resuscitation techniques to reverse clinical death, however Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2, has led to increasingly ire quent observations of a pathological condition occurring in patients who remain unconscious, involving multiple organ inEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
jury or failure following repertiision after prolonged cardiac arrest.The concept of postresuscitarion disease as a unique and new nosological entity PartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2mbination of severe circulatory hypoxia with the unintended sequelae of measures used for resuscitation.On the basis of the wide variety of ischemic/hypoxic mechanisms that can trigger its development, the disease was redefined by Safar as a syndrome in which pathogenetic processes triggered by card Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2iac arrest were exacerbated by reperfusion, causing damage to the brain and other organs, the complex interactions of which combine to determine overaEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
ll outcome (see early experimental findings summary).67The evidence of features common to the postresuscitation syndrome and multiple organ dysfunctioPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2 damage directly induced by ischemia during cardiac arrest."Iwo landmark studies, showing that mild therapeutic hypothermia started after reperfusion can improve recov cry after cardiac arrest, confirm that outcome is deter mined not only hy events occurring during arrest and CPR but also by pathoge Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2netic processes continuing after reperfusion.910Recent reports confirm the occurrence of a "sepsis-like syndrome" after resuscitation from cardiac arrEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
est, although the mechanistic relationship to the direct damage induced by ischemia during cardiac arrest lias yet to be clarified.CaniiacArml: Vie ScPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2ed by Cambridge University Press. 0 Cambridge University Press, 2007.817818Erga L. CcrchiariEarly Experimental FindingsNegovsky1’’and his group of Russian investigators pioneered the concept of post resuscitation disease as a unique nosological entity, caused by the combination of severe hypoxia and Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2 resuscitation, on the basis of hundreds of experimental observations that fall into three groups:1Phasic Pattern of Postresuscitation RecoveryIndepenEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
dent of the type of insult, alterations in cerebral and extracerebral organs occur starting with reperfusion and developing over time.From instill to PartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2crease levels of biologically active substances and prostaglandin derivatives; alterations of the immune system (increased bactericidal activity, depressed reticuloendothelial system, and hyperreactivity of B- and T-lymphocytes), and toxic factors In the blood (peptide fraction 800 to 2000 Daltonsan Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2d endotoxin secondary to gram-negative bacteremia).From 10 to 24 hours postinsult: normalization of cardiovascular variables and progression of metaboEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
lic derangements ensue. During this time. 50% of deaths occur as a result of recurrent cardiac arrest.From I to 3 days postinsult: stable cardiovasculPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2e than 3 days postinsult): characterized by the prevalence of localized or generalized Infection that represents the major cause of delayed deaths. The degree of cerebral and extracerebral organ derangements Is reported to be more severe and prolonged the longer the duration of the hypoxic-ischemic Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2insult.2Interactions between Cerebral and Extracerebral Postischernic Damage on OutcomeThe severity of systemic and hemodynamic derangements after 20Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
minutes of isolated brain ischemia is comparable to that recorded after only 12-15 minutes of total circulatory arrest with ventricular fibrillation, PartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2gulation.Cerebral function recovers better after bloodless global brain ischemia than after the same duration of circulatory arrest from ventricular fibrillation, leading to the conclusion that extracerebral factors account for about half of the pathological findings in the brain Induced by cardiac Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2arrest.3Benefical Effect of Trials with Detoxification TechniquesA series of trials aimed at removing toxins and normalizing homeostasis by various deEbook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
toxification techniques showed that all the techniques can improve neurological recovery anti survival compared with concurrent controls: cross-circulPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2hy donor dog, aided by an extracorporeal circulation system.Safar and his group in Pittsburgh, in parallel with - but subsequent to - the Russian experimental work, confirmed that extracerebral organ dysfunction may hamper cerebral recovery following resuscitation from cardiac arrest, based on the o Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2bservations that (a) cerebral function after Isolated global brain Ischemia recovers better than after comparable durations of total body ischemia1*14Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2
and (b) the use of cardiopulmonary bypass for resuscitation and for short-term postresuscitation assistance improves myocardial performance after weaPartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iai Ebook Cardiac arrest - The science and practice of resuscitation medicine (2nd edition): Part 2 of increasing durations was studied in animal experimental models:,f’'7>',‘,!'PartvPostresuscitation disease and its care47Postresuscitation syndromeErga L. Cerchiaridepartment of Anaesthesia and cntical Care.OqirtLdc- v.«|i|iaiGọi ngay
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