Ebook Dermatology at a glance: Part 2
➤ Gửi thông báo lỗi ⚠️ Báo cáo tài liệu vi phạmNội dung chi tiết: Ebook Dermatology at a glance: Part 2
Ebook Dermatology at a glance: Part 2
Tropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2he human skin, especially via sites in contact with contaminated soil (eg. feet, buttocks). Patients present with an extremely itchy, erythematous, serpiginous track that extends over days due to larval movement (Figure 22.1). The track consists of a combination of papules, vesicles or blisters. Dia Ebook Dermatology at a glance: Part 2gnosis is made clinically. The condition is self-limiting as larvae die within 6-8 weeks of penetrating the skin Treatment of limited disease is withEbook Dermatology at a glance: Part 2
topical 10% thiabendazole cream for 1- 2 weeks; if widespread disease, oral albendazole or ivermectin.Leishmaniasis J/Caused by different species of tTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2 Incubation period ranges from weeks to a year. There are three main types:1Cutaneous is the most common form. Patients present with one or more painless ulcers (termed tropical ulcers; Figure 22.2) on exposed skin, often the face or limb, which heals over months with scarring. Regional lymphadenopa Ebook Dermatology at a glance: Part 2thy may be present.2Mucocutaneous form, occurs months or years after healing of a cutaneous leishmaniasis lesion. May lead to partial or complete destEbook Dermatology at a glance: Part 2
ruction of mucous membranes (e.g. nasopharynx).3Visceral form (kala azar) is the most severe, due to visceral organ involvement. Patients present withTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2 infection.About 90% of visceral leishmaniasis occurs in the Indian subcontinent. Sudan. Ethiopia and Brazil; 90% of cutaneous leishmaniasis is in Afghanistan. Saudi Arabia and South America.Diagnosis is confirmed by histology and culture of scrapings and/or biopsy of affected skin (cutaneous form): Ebook Dermatology at a glance: Part 2 by light microscopic examination or culture of parasites from splenic or bone marrow aspirates (visceral form). Polymerase chain reaction (PC’R) of tEbook Dermatology at a glance: Part 2
issue samples is used to identify the disease-causing species of leishmaniasis, which influences treatment choice.Treatment ts with pentavalent antimoTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2thing.LeprosyLeprosy is a notifiable disease in the UK. caused by Mycobacterium leprae, an intracellular bacterium with a predilection for skin, peripheral nerves, respiratory mucosa and eyes. The prevalence is falling, but it is still endemic in India. Africa and South America.M. leprae causes a ch Ebook Dermatology at a glance: Part 2ronic granulomatous reaction leading to:•Skin: anaesthetic hypopigmented macules ± erythematous plaques (Figure 22.3a.b).•Peripheral nenes: enlarged pEbook Dermatology at a glance: Part 2
eripheral nerves (Figure 22.3a) and peripheral neuropathy.•Eyes: blindness due to direct bacillary infiltration and neuropathy resulting in diminishedTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2 to several years. There arc two main subtypes:1Tuberculoid: strong immune response to M. leprae, few skin lesions, low bacterial load (paucibacillary)2l.epromatous: poor immune response to M. leprae, severe disease, with multiple skin lesions and high bacterial load (miihibacillary).Diagnosis is by Ebook Dermatology at a glance: Part 2 finding acid-fast bacilli on Ztehl-Ncclson staining in smears and/or biopsies from affected skin or other tissue. Serological tests can detect M. lepEbook Dermatology at a glance: Part 2
rae antigens or antibodies. Treatment is with rifampicin, dapsone and dofazamine. depending on disease subtype. Erythema nodosum leprosum is an immuneTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2is and leprosy.► Warning•Cutaneous leishmaniasis may progress to mucocutaneous form, leading to mucous membrane destruction.•Untreated visceral leishmaniasis is potentially fatal.Darmslofogy at a Gfisnce. First Edition. Mahbub M.U. Chowdhury. Ruwart p. Katuqampola. and Andrew Y. Finlay.o 2013 John W Ebook Dermatology at a glance: Part 2iley « Sons. Ltd. Putnshsd 2013 by John Wroy « Sons. Ltd. 49The red facefable 23.1 Specific questions and history•Where did the eruption start?•Any hiEbook Dermatology at a glance: Part 2
story of previous eidn dowse or family history of ekin dowse?•Is thio facial eruption Itchy?•Is this facial eruption ecaỹ?•Any fluohrg or increased reTropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into th Ebook Dermatology at a glance: Part 2or tredneeo (anaemia)?Tropical skin diseaseCutaneous larva migransCaused by hookworm larvae in soil contaminated by animal faeces. Tlie larvae penetrate and migrate into thGọi ngay
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