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Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

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Nội dung chi tiết: Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

Chapter 11TCA Cycle and OxidativePhosphorylationThis chapter Conklins questions on (he TCA cycle and oxidative phosphorylation, including questions in

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2ntegrated w ith other aspects of metabolism. Metabolic diseases affecting aspects of the TCA cycle and oxidative phosphorylation arc also covered in (

his chapter.QUESTIONSSelect the single best answer.A chronic alcoholic, while out on a binge. became very confused and forgetful. The police found the Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

man and brought him to tile emergency department. Upon examination. he displayed nystagmus and ataxia. Which enzyme is displaying reduced activity tn

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

his brain under these conditions?(A)Glyceraklehyde-3-phosphate dehydrogenase(B)Isocitrate dehydrogenase(C)a-kcloglutaratc dehydrogenase(D)Succinate d

Chapter 11TCA Cycle and OxidativePhosphorylationThis chapter Conklins questions on (he TCA cycle and oxidative phosphorylation, including questions in

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2 high-energy bonds formed?(A)6(B)8(C)10(D)12(E)H3Ethanol ingestion is incapable of supplying carbons for gluconeogenesis. This is due to which of the

following?(A)Ethanol is converted to acetone, and the carbons are lost during exhalation(B)Ethanol is lost directly in the urine(C)Ethanol cannot ente Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

r the liver, where gluconeogenesis predominantly occurs(D)Ethanols carbons are lost as carbon dioxide before a gluconeogenic precursor can be generate

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

d(E)Ethanol IS converted to lysine, which IS strictly a kclogcnic amino acid4A family that had previously had 3 newborn boy die of a metabolic disease

Chapter 11TCA Cycle and OxidativePhosphorylationThis chapter Conklins questions on (he TCA cycle and oxidative phosphorylation, including questions in

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2analysis indicated extremely high levels of lactic acid. Analysis of cerebrospinal fluid showed elevated lactate and pyruvate. Hypcralanincmia was als

o observed. The child died within 5 days of birth. The biochemical defect in this child is most likely which of the following?(A)The E1 subunit of pyr Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

uvate dehydrogenase(B)The E2 subunit of pyruvate dehydrogenase(C)The E3 subunit of pyruvate dehydrogenase(D)Citrate synthase(E)Malate dehydrogenase5A

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

3-month-old girl developed lactic acidemia. Blood analysis also indicated elevated levels of pyruvate. a-ketoglutarate. and branched-cham amino acids.

Chapter 11TCA Cycle and OxidativePhosphorylationThis chapter Conklins questions on (he TCA cycle and oxidative phosphorylation, including questions in

Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2 the following proteins would lead to this clinical finding?(A)The E1 subunit of pyruvate dehydrogenase(B)The E2 subunit of pyruvate dehydrogenase(C)T

he E3 subunit of pyruvate dehydrogenase(D)Citrate synthase(E)Malate dehydrogenase89 Ebook Lippincott''s illustrated Qamp;A review of biochemistry: Part 2

Chapter 11TCA Cycle and OxidativePhosphorylationThis chapter Conklins questions on (he TCA cycle and oxidative phosphorylation, including questions in

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